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Black tea may help with weight loss, too

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Researchers have demonstrated for the first time that black tea may promote weight loss and other health benefits by changing bacteria in the gut. In a study of mice, the scientists showed that black tea alters energy metabolism in the liver by changing gut metabolites. The study found that both black and green tea changed the ratio of intestinal bacteria in the animals: The percentage of bacteria associated with obesity decreased, while bacteria associated with lean body mass increased.

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Possible therapeutic target for regulating body weight

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A new study published online in The FASEB Journal reveals a novel gene involved in maintaining body weight. Specifically, the study suggests that GTRAP3-18 interacts with pro-opiomelanocortin (POMC) in the hypothalamus to regulate food intake and blood glucose levels. Inhibiting the interaction between GTRAP3-18 and POMC might be a strategy for treating leptin/insulin resistance in patients with obesity and/or type 2 Diabetes. Researchers have analyzed a group of mice defective in the GTRAP3-18 gene. The GTRAP3-18-deficient mice were lean as compared with wild type mice. The leanness was due to neither increased locomotive activity nor basal metabolism, but rather a dysregulation of feeding behavior, or hypophagia. The GTRAP3-18-deficient mice also displayed hypoglycemia.

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Too much sugar? Even ‘healthy people’ are at risk of developing heart disease

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Healthy people who consume high levels of sugar are at an increased risk of developing cardiovascular disease. A ground-breaking study from the University of Surrey found that a subject group of otherwise healthy men had increased levels of fat in their blood and fat stored in their livers after they had consumed a high sugar diet. The study looked at two groups of men with either high or low levels of liver fat, and fed them a high or low sugar diet to find out if the amount of liver fat influences the impact of sugar on their cardiovascular health. The low sugar diet contained no more than 140 calories a day worth of sugar — an amount close to the recommended intake — while the high sugar diet contained 650 calories worth. After 12 weeks on the high sugar diet, the men with a high level of liver fat — a condition known as non-alcoholic fatty liver disease (NAFLD) — showed changes in their fat metabolism that are associated with an increased risk of cardiovascular disease, heart attacks and strokes.

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Obesity gene no barrier to weight loss, study shows

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Diet, exercise and drug-based approaches to losing weight can be widely beneficial, even if some people may have a greater risk of piling on the pounds due to their genetics. In short, your DNA is not a barrier to weight loss. While many genes are believed to affect body weight, a particular version of the so-called FTO gene shows one of the strongest associations with fat gain. Those carrying two copies of the genetic variant – about 16% of the population – are on average 3kg heavier than those without, and 1.7 times more likely to be obese. While it is not known exactly how the genetic variant promotes weight gain, it is believed to increase the appeal of high-calorie foods and reduce the feeling of fullness after a meal. But whether it also affects efforts to shed pounds has been a matter of debate.

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Your brain, not your stomach, tells you when to stop eating

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Cholecystokinin:

A hormone produced by your digestive system, called cholecystokinin, signals your brain when you’ve eaten. More is released in a large meal and less from a small snack. When you are dieting to lose weight, your stomach counterbalances the reduced food intake by cutting back on the amount of this hormone it will release, encouraging you to eat more and go off your diet. Loading up on fiber will help you circumvent this built-in survival tool.

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The food industry is behaving as the tobacco industry

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Increasingly, the question of what we eat and how it affects our health is a subject that is important not just to those concerned about nutrition but to environmentalists. Food Fight, Brownell has recently become interested in the connections between obesity, the environment, and hunger, believing that sustainably growing and producing more nutritious foods can help solve each of these challenges. this is a perfect repeat of what tobacco did for many years. They said smoking doesn’t cause lung cancer. There is not definitive evidence. There aren’t good-enough studies. It’s junk science. It’s just the advocates out to get us. And then they denied that second-hand smoke was killing people. They denied that nicotine was addictive. You can go on and on and on. Well, so here comes a (food) study that’s pretty persuasive. It certainly supports other studies showing a link between fast food consumption and obesity, and what did they do? They trashed the science. They deny it’s the case. In all likelihood, they will pay scientists who they know to produce results favourable to them to disprove this finding.

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The body’s own fat-metabolism protects against the harmful effects of sugar

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Researchers have discovered that the fat-metabolism in the cells takes place simultaneously with a detoxification of the harmful substances from the blood sugar, which can avert the damage that can in turn lead to age-related diseases such as diabetes, Alzheimer’s and cancer. This indicates that we have a detoxification system which we were not previously aware of.

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