How fat tissue shunts energy to tumors

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The loss of p62 curtails energy-consuming activities in fat cells, leaving more nutrients available for tumor growth

Source: Sanford-Burnham Prebys Medical Discovery Institute

Summary: Researchers recently discovered that that inactivation of a protein called p62 in fat cells fuels aggressive, metastatic prostate cancer in mice. The findings suggest that mTOR inhibitors currently used to treat a wide range of cancers may have the unintended consequence of shutting down fat tissue metabolism and fuelling tumor growth.

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